Lipoxygenases and their Products by Stanley T. Crooke

By Stanley T. Crooke

Lipoxygenases are enzymes curious about the conversion of arichidonic acid to leukotrienes and lipoxins. those serve a number of very important features and feature been implicated in quite a lot of human illnesses. This e-book seems on the constitution, pharmacology, and mobile mechanisms of motion of lipoxygenases and their metabolic items.

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Foley, J. , and Wasserman, M. A. (1987). Inhibition of leukotriene D4-induced coronary vasoconstriction by leukotriene antagonists in the anesthetized dog. J. Pharmacol. Exper. Therap. 241, 174-180. 121. Krell, R. , and Bryan, D. (1981). Contraction of isolated airway smooth muscle by synthetic leukotrienes C4 and D4. Prostaglandins 22, 387-409. 122. Krell, R. , Snyder, D. , Giles, R. , Lee, Y. , Bernstein, P. , Brown, F. , and Hesp, B. (1986). ICI 198,615, a novel peptidoleukotriene (LT) receptor antag­ onist: In vivo pharmacology.

A. (1983). Immunologie and ionophore-induced generation of leukotriene B4 from mouse bone marrow-derived mast cells. J. Immunol. 130, 1885-1890. , Hill, R. , Watkins, W. , Petkau, A. , and Zapol, W. M. (1983). Effects of leukotrienes B4 and C4 on coronary circulation and myocardial contractility. Surgery 94, 267-275. 1. Pharmacology and Pathophysiology of 5-Lipoxygenase Products 45 142. , Hill, R. , Watkins, W. , Petkau, A. , and Zapol, W. M. (1982). Leukotriene D4: a potent coronary artery vasoconstrictor associated with impaired ventricular contraction.

J. Pharmacol. Exp. Therap. 235, 470-474. 1. Pharmacology and Pathophysiology of 5-Lipoxygenase Products 39 44. Cristol, J. , and Sirois, P. (1988). Comparative activity of leukotriene D4, 5,6dihydroxyeicosatetranoic acid and lipoxin A on guinea pig lung parenchyma and ileum smooth muscle. Res. Comm. Chem. Path. Pharmacol. 59, 423-426. 45. Dahlen, S. , Arfors, K. , and Samuelsson, B. (1981). Leukotrienes promote plasma leakage and leuko­ cyte adhesion in postcapillary venules: in vivo effects with relevance to the acute inflammatory response.

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