By Hamid Sayar
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Additional resources for Myelodysplastic Syndromes: From Pathogenesis to Diagnosis and Therapy (Recent Advances in Hematology Research)
Select FCI abnormalities in MDS bone marrow samples. A. Increased numbers of blasts. The cell populations include erythroid cells (in black), blasts (in red), granulocytes (in navy blue ), lymphocytes (in blue) and monocytes (in green). B. Prominent CD38-CD34+ blast population in MDS sample. C. Aberrant expression of CD56 in CD34+ blasts. D. Abnormal expression of CD7 in CD34+ blasts. E. Asynchronous expression of maturing marker CD11b in blasts. F. Maturing granulocytes show loss of CD10 antigen.
1992. 16(1): p. 43-6.  Christiansen, D. , Andersen, M. , Mutations of AML1 are common in therapy-related myelodysplasia following therapy with alkylating agents and are significantly associated with deletion or loss of chromosome arm 7q and with subsequent leukemic transformation. Blood, 2004. 104(5): p. 1474-81. , Hyperactivation of the RAS signaling pathway in myelodysplastic syndrome with AML1/RUNX1 point mutations. Leukemia, 2006. 20(4): p. 635-44. , Andersen, M. , Andersen, M. , Genetic pathways in the pathogenesis of therapy-related myelodysplasia and acute myeloid leukemia.
Epilepsia, 1993. 34(6): p. 1098-101. , Implications of discrepancy in morphologic diagnosis of myelodysplastic syndrome between referral and tertiary care centers. Blood. 118(17): p. 4690-3. , Myelodysplastic syndromes: 2011 update on diagnosis, riskstratification, and management. Am. J. Hematol. 86(6): p. 490-8.  Wang, S. , Diagnosis of myelodysplastic syndromes in cytopenic patients. Hematol. Oncol. Clin. North Am. 25(5): p. 1085-110, vii. , Clinical significance of phenotypic features of blasts in patients with myelodysplastic syndrome.