By Dale Dubin
The reader's fast assimilation of scientific recommendations is the main to the ongoing luck of this best-selling e-book. A caption explains the idea that illustrated on every one web page, and some easy sentences toughen the idea that with interactive (programmed) studying, which hyperlinks to the next web page. Dr. Dubin's mild and exciting sort, recognized world-wide, makes studying stress-free. perform twelve-lead tracings on the finish identify self-confidence, and summarised reference sheets with examples (designed to be copied) offer a superb assessment. This paintings is so much lately up to date in February 2006.
The illustrations in quick Interpretation of EKG's use actual EKG tracings (not simulations), so what you spot, is what you are going to event professionally. scholars within the clinical sciences, frequently intimidated by means of electrocardiography, welcome this learning-by-understanding process. Memorizing styles of EKG's has a depressing half-life. utilizing wisdom anchored in knowing, readers are happy that, in a refined demeanour, in addition they know about cardiac body structure, pathology of the center, and care of sufferers. The culture persists.
The hottest programmed texts owe their good fortune to reader/student suggestions. No writer has been capable of expect pages that may be tricky for neophytes to understand. So, reader enter has develop into instrumental within the evolution of such texts. New explanatory textual content is extra with each one new printing to ease the training approach via parts of highbrow turbulence. the writer courses your development for swift, seamless comprehension.
Through the years, modern "programmed instruction" has advanced into "interactive learning" wherein the reader actively participates in his personal studying procedure. scientific schooling needn't be an oppressively critical job. over the years Dr. Dubin's humorousness has been many times woven into the conversation with the reader, lightening the tensions of research. Many authors have mimicked his pleasant, easy-going style.
Rapid Interpretation of EKG's employs interactive studying to accomplish greatest figuring out.
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Extra resources for Rapid Interpretation of EKG's (6th Edition)
Qxd 26 3/30/09 4:41 PM Page 26 IVUS MADE EASY Figure 24 Plaque (atheroma) measurements In this figure, two highly stenotic lesion sites are shown. Importantly, the size of the plaque and the atheroma burden of the lesion in the right panels are significantly larger, despite the similar lumen dimensions. Maximum and minimum plaque thickness The largest and shortest distance from the intimal leading edge to the EEM border along any line passing through the center of the lumen, respectively. The circumferential extent of disease is commonly classified by determining whether abnormal intimal thickening is present throughout the 360° arterial circumference.
The difference in size and shape of the vessel is illustrated in the graphics below. qxd 20 3/30/09 4:37 PM Page 20 IVUS MADE EASY Figure 20 Muscle bridge The cyclical pattern of artery size is reversed at the site of muscle bridges. Here, the artery size reaches a minimum during systole (right), caused by contraction of the muscle surrounding the vessel. practice this is often not the case, and both transducer obliquity and vessel curvature can introduce an elliptical image distortion. Transducer obliquity is especially important in large vessels, and can result in an overestimation of dimensions and a reduction in image quality36.
Qxd 3/30/09 4:43 PM Page 43 PLAQUE (ATHEROMA) MORPHOLOGY 43 and acute coronary syndromes76–78. The pathophysiologic processes responsible for plaque destabilization are incompletely understood, but systemic, and in particular inflammatory, triggers play an important role73,79–99. Conversely, fibrotic changes may increase internal plaque resistance to rupture100. The balance between inflammation and fibrosis may be an important determinant of plaque progression/regression and vulnerability101. The role of systemic triggers is supported by the recent observation that plaque destabilization in patients presenting with acute coronary syndromes is characterized by the diffuse development of multiple vulnerable lesions simultaneously78–84.