The Fetal Matrix: Evolution, Development and Disease by Peter Gluckman, Mark Hanson

By Peter Gluckman, Mark Hanson

New discoveries point out that an important interactions in identifying our destiny happen sooner than beginning. those tactics are an evolutionary echo of mechanisms which allowed our ancestors to outlive as hunter-gatherers. of the world's best pioneering gurus demonstrate fascinating insights right into a quickly rising box. They recommend new methods of shielding the future health of the fetus, child and grownup and canopy very important triggers for plenty of rising ailments comparable to weight problems, diabetes and middle ailment.

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Extra resources for The Fetal Matrix: Evolution, Development and Disease

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Exposure to certain drugs as an embryo will lead to permanent developmental changes; exposure to the same drug as an adult will only have effects while it is being administered. For example, stilboestrol is a synthetic form of oestrogen that was tried in the 1960s as a treatment for recurrent miscarriage. It can be safely used as a replacement oestrogen in non-pregnant adult women but if a female fetus is exposed to it, that fetus will have a high risk of developing a rare form of vaginal cancer in adolescent or adult life.

Fava beans) and the genotype (the faulty G6PD gene). This is a dramatic example of what is likely to be the most common way in which genetic factors predispose to disease – they change the way in which environmental factors impact on the function of the body. Genes have been related to many common diseases such as diabetes and heart disease. Usually there are multiple genes involved and the genes are not in themselves causal. They create a risk situation in which a particular set of genetically determined changes in body function, together with environmental factors, creates disease.

The dialogue between the fetus and its placenta, effectively unknown to the mother, is very vigorous. The placenta is not only about nutrient transfer; it is also an important source of hormones that influence both maternal and fetal function. From about the seventh week onwards the human placenta will make the hormone chorionic gonadotropin, which enters the fetus and stimulates the developing testis of the male to make testosterone – this in turn is a critical step in the development of the external genitalia of the male.

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